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Molecular Autism

來源: 樹人論文網(wǎng) 瀏覽次數(shù):250次
所屬分區(qū):2區(qū)
ISSN:2040-2392
影響因子:5.712
是否開源:Yes
年文章量:65
研究方向:醫(yī)學
官網(wǎng)地址:http://molecularautism.biomedcentral.com/
投稿地址:https://www.editorialmanager.com/mola/default.aspx

Molecular Autism雜志中文介紹

《分子自閉癥》是一本經(jīng)過同行評審的開放獲取期刊,出版高質(zhì)量的基礎、轉化和臨床研究,與自閉癥的病因學、病理生物學或治療以及相關的神經(jīng)發(fā)育條件有關。鼓勵包括跨級別集成的研究。分子自閉癥發(fā)表經(jīng)驗研究、評論和簡短的交流。我們鼓勵來自不同領域的投稿,包括(但不限于)遺傳學、分子神經(jīng)生物學、神經(jīng)病理學、神經(jīng)成像、認知神經(jīng)科學、流行病學和生物標志物的發(fā)現(xiàn)。《分子自閉癥》雜志還發(fā)表了關于篩查、診斷和分類的文章,包括考慮進行分組以改進我們對基本機制的理解的文章。干預研究也很受歡迎,尤其是在考慮揭示因果機制時。雖然主要關注自閉癥譜系障礙(包括阿斯伯格綜合癥),但范圍包括對相關神經(jīng)發(fā)育障礙的分子研究,如特定語言障礙、運動障礙和特定或一般發(fā)育遲緩;和相關的醫(yī)學癥狀,如脆性X綜合征,結節(jié)性硬化癥,和勒特綜合癥。《分子自閉癥》也考慮沒有分子數(shù)據(jù)的文章,但從長遠來看,這些文章可能會縮小自閉癥患者從分子到行為之間的差距。該雜志歡迎來自這一復雜事件鏈的兩端的報告和對優(yōu)秀科學的評論:要么從分子上,要么從行為上。報告和審查可以是基本的和/或翻譯的。請注意,由于我們很少接受未經(jīng)請求的評審,如果潛在評審的作者認為某個重要主題沒有得到充分的評審,并且在理想情況下,他們建議進行系統(tǒng)評審,我們強烈建議他們使用提交前查詢系統(tǒng)。

Molecular Autism雜志英文介紹

Molecular Autism is a peer-reviewed, open access journal that publishes high-quality basic, translational and clinical research that has relevance to the etiology, pathobiology, or treatment of autism and related neurodevelopmental conditions. Research that includes integration across levels is encouraged. Molecular Autism publishes empirical studies, reviews, and brief communications.We encourage submissions from a range of fields including (but not restricted to) genetics, molecular neurobiology, neuropathology, neuroimaging, cognitive neuroscience, epidemiology, and biomarker discovery. Molecular Autism also publishes articles on screening, diagnosis and classification, including articles that consider subgrouping to refine our understanding of basic mechanisms. Intervention studies are also welcome, especially when considered with respect to revealing causal mechanisms.Although the primary focus is on conditions on the autism spectrum (including Asperger syndrome), the scope encompasses molecular research into related neurodevelopmental conditions such as specific language impairment, dyspraxia, and specific or general developmental delays; and into related medical syndromes such as fragile X syndrome, tuberous sclerosis, and Rett syndrome.Molecular Autism also considers articles with no molecular data but which, in the long-term, may close the gap from molecule to behavior in autism. The journal welcomes reports and reviews of good science that proceed from either end of this complex chain of events: from molecule upwards, or from behavior downwards. Reports and reviews can be basic and/or translational. Note that, because we rarely accept unsolicited reviews, we strongly encourage authors of potential reviews to make use of the presubmission inquiry system if they feel that an important topic has not been adequately reviewed and where, ideally, they are proposing a systematic review.

Molecular Autism影響因子

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